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Sickle Cell Disease African Killer: Biologists Alternative

Otoikhian C S O and Okoror L E
Department of Food Science, Periyar University, Salem-636 011, Tamil Nadu, India.

Abstract—This seminar paper is done towards the elimination of sickle cell disease in our environment. Sickle cell anemia is an inherited disease of the red blood cells in which the red blood cell become sickle-shaped (crescent shaped) and has difficulty passing through small blood vessels. This process produces periodic episodes of pain and ultimately can damage tissues and vital organs and lead to other serious medical problems. Symptoms include fatigue, joint and abdominal pain, irritability, yellow discoloration of the skin and eyes, leg sores, gum disease, frequent respiratory infections, blindness later in life, and periods of prolonged, sometimes painful erections in males. People with sickle cell anemia can have episodes of severe pain in the arms, legs, chest, and abdomen that may be accompanied by fever, nausea, and difficulty breathing. These symptoms occur only in people who inherit copies of the sickle cell gene from both parents. Regular health maintenance is critical for people with sickle cell anemia. Today, proper nutrition, good hygiene, bed rest, protection against infection, regular visits to physician, many people with sickle cell anemia are in reasonably good health much of the time and living productive lives. Before starting a family, a simple blood test can reveal if one or both parents is a carrier. In conclusion, before starting a family, a simple blood test should be done because a child that receives the defective gene from both parents develops the disease. Therefore, marriage between carriers should be discouraged because sickle cell is better not produced than experienced.

Index Terms—Sickle cell anemia, African killer, Red blood cells

Cite: Otoikhian C S O and Okoror L E, "Sickle Cell Disease African Killer: Biologists Alternative," International Journal of Pharma Medicine and Biological Sciences, Vol. 1, No. 2, pp. 232-245, October 2012.
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